Mitochondrial dysfunction

Lactoferrin decreases LPS-induced mitochondrial dysfunction in cultured cells and in animal endotoxemia model

Immune response / Oxidative Stress / Mitochondria / DNA damage / Innate immunity / Lipopolysaccharide / Humans / Reactive Oxygen Species / Mice / Animals / Siderophores / P-glycoprotein / Iron / Innate / Hydrogen Peroxide / Mitochondrial dysfunction / Lipopolysaccharides / ENDOTOXEMIA / Epithelial cells / Monocytes / Oxidative Damage / Deferoxamine / Lipopolysaccharide / Humans / Reactive Oxygen Species / Mice / Animals / Siderophores / P-glycoprotein / Iron / Innate / Hydrogen Peroxide / Mitochondrial dysfunction / Lipopolysaccharides / ENDOTOXEMIA / Epithelial cells / Monocytes / Oxidative Damage / Deferoxamine

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Lactoferrin decreases LPS-induced mitochondrial dysfunction in cultured cells and in animal endotoxemia model

Immune response / Oxidative Stress / Mitochondria / DNA damage / Innate immunity / Lipopolysaccharide / Humans / Reactive Oxygen Species / Mice / Animals / Siderophores / P-glycoprotein / Iron / Innate / Hydrogen Peroxide / Mitochondrial dysfunction / Lipopolysaccharides / ENDOTOXEMIA / Epithelial cells / Monocytes / Oxidative Damage / Deferoxamine / Lipopolysaccharide / Humans / Reactive Oxygen Species / Mice / Animals / Siderophores / P-glycoprotein / Iron / Innate / Hydrogen Peroxide / Mitochondrial dysfunction / Lipopolysaccharides / ENDOTOXEMIA / Epithelial cells / Monocytes / Oxidative Damage / Deferoxamine

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Mitochondrial control of autophagic lysosomal pathway in Alzheimer\'s disease

Psychology / Oxidative Stress / Mitochondria / Autophagy / Energy Metabolism / Brain / Humans / Protein Aggregation / Clinical Sciences / Mitochondrial dysfunction / Experimental / Experimental Neurology / Lysosomes / Alzheimer Disease / Cell Survival / Neurosciences / Macroautophagy / Brain / Humans / Protein Aggregation / Clinical Sciences / Mitochondrial dysfunction / Experimental / Experimental Neurology / Lysosomes / Alzheimer Disease / Cell Survival / Neurosciences / Macroautophagy

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Lactoferrin decreases LPS-induced mitochondrial dysfunction in cultured cells and in animal endotoxemia model

Immune response / Oxidative Stress / Mitochondria / DNA damage / Innate immunity / Lipopolysaccharide / Humans / Reactive Oxygen Species / Mice / Animals / Siderophores / P-glycoprotein / Iron / Innate / Hydrogen Peroxide / Mitochondrial dysfunction / Lipopolysaccharides / ENDOTOXEMIA / Epithelial cells / Monocytes / Oxidative Damage / Deferoxamine / Lipopolysaccharide / Humans / Reactive Oxygen Species / Mice / Animals / Siderophores / P-glycoprotein / Iron / Innate / Hydrogen Peroxide / Mitochondrial dysfunction / Lipopolysaccharides / ENDOTOXEMIA / Epithelial cells / Monocytes / Oxidative Damage / Deferoxamine

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Normal Serum Alanine Concentration Differentiates Transient Neonatal Lactic Acidemia from an Inborn Error of Energy Metabolism

Decision Making / Energy Metabolism / Mitochondrial DNA / Humans / Mutation / Inborn errors of metabolism / Differential Diagnosis / Clinical Sciences / Newborn Infant / Mitochondrial dysfunction / Mitochondrial Diseases / Pilot study / Oxidative phosphorylation / Alanine / Pilot Projects / Lactic Acid / Inborn errors of metabolism / Differential Diagnosis / Clinical Sciences / Newborn Infant / Mitochondrial dysfunction / Mitochondrial Diseases / Pilot study / Oxidative phosphorylation / Alanine / Pilot Projects / Lactic Acid

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Knockdown of Cytosolic Glutaredoxin 1 Leads to Loss of Mitochondrial Membrane Potential: Implication in Neurodegenerative Diseases

Immunohistochemistry / Mitochondria / Multidisciplinary / Neurodegenerative Diseases / Antioxidants / Humans / Reactive Oxygen Species / Polymerase Chain Reaction / Animal Model / PLoS one / Mitochondrial dysfunction / Neurodegenerative Disease / Matrix Metalloproteinases / Mitochondrial Permeability Transition Pore / Base Sequence / Mitochondrial Membrane Potential / Outer Membrane Protein / Nucleotides / Down-Regulation / Humans / Reactive Oxygen Species / Polymerase Chain Reaction / Animal Model / PLoS one / Mitochondrial dysfunction / Neurodegenerative Disease / Matrix Metalloproteinases / Mitochondrial Permeability Transition Pore / Base Sequence / Mitochondrial Membrane Potential / Outer Membrane Protein / Nucleotides / Down-Regulation

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Glicación de proteínas mitocondriales, estrés oxidativo y envejecimiento

Aging / Oxidative Stress / Mitochondria / Protein Structure and Function / Glycation / Humans / Animals / Glycosylation / Mitochondrial dysfunction / Chemical Modification / Humans / Animals / Glycosylation / Mitochondrial dysfunction / Chemical Modification

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Lactoferrin decreases LPS-induced mitochondrial dysfunction in cultured cells and in animal endotoxemia model

Immune response / Oxidative Stress / Mitochondria / DNA damage / Innate immunity / Lipopolysaccharide / Humans / Reactive Oxygen Species / Mice / Animals / Siderophores / P-glycoprotein / Iron / Innate / Hydrogen Peroxide / Mitochondrial dysfunction / Lipopolysaccharides / ENDOTOXEMIA / Epithelial cells / Monocytes / Oxidative Damage / Lipopolysaccharide / Humans / Reactive Oxygen Species / Mice / Animals / Siderophores / P-glycoprotein / Iron / Innate / Hydrogen Peroxide / Mitochondrial dysfunction / Lipopolysaccharides / ENDOTOXEMIA / Epithelial cells / Monocytes / Oxidative Damage

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Mitochondrial dysfunction promoted by Porphyromonas gingivalis lipopolysaccharide as a possible link between cardiovascular disease and periodontitis

Oxidative Stress / Mitochondria / Apoptosis / Cardiovascular disease / Free Radical / Inflammatory Immune Response / Lipopolysaccharide / Humans / Reactive Oxygen Species / Inflammatory disease / Mitochondrial dysfunction / Middle Aged / Lipopolysaccharides / Adult / Protein Expression / Periodontitis / Cardiovascular Diseases / Human Fibroblasts / Mitochondrial Membrane Potential / Citrate Synthase / Biochemistry and cell biology / PORPHYROMONAS GINGIVALIS / Inflammatory Immune Response / Lipopolysaccharide / Humans / Reactive Oxygen Species / Inflammatory disease / Mitochondrial dysfunction / Middle Aged / Lipopolysaccharides / Adult / Protein Expression / Periodontitis / Cardiovascular Diseases / Human Fibroblasts / Mitochondrial Membrane Potential / Citrate Synthase / Biochemistry and cell biology / PORPHYROMONAS GINGIVALIS

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Mitochondrial DNA damage as a potential mechanism for age-related macular degeneration.

Aging / Retinal Pigment Epithelium / Mitochondrial DNA / Age-related Macular Degeneration / Mitochondrial dysfunction

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Mitochondrial dysfunction promoted by Porphyromonas gingivalis lipopolysaccharide as a possible link between cardiovascular disease and periodontitis

Oxidative Stress / Mitochondria / Apoptosis / Cardiovascular disease / Free Radical / Inflammatory Immune Response / Lipopolysaccharide / Humans / Reactive Oxygen Species / Inflammatory disease / Mitochondrial dysfunction / Middle Aged / Lipopolysaccharides / Adult / Protein Expression / Periodontitis / Cardiovascular Diseases / Human Fibroblasts / Mitochondrial Membrane Potential / Citrate Synthase / Biochemistry and cell biology / PORPHYROMONAS GINGIVALIS / Inflammatory Immune Response / Lipopolysaccharide / Humans / Reactive Oxygen Species / Inflammatory disease / Mitochondrial dysfunction / Middle Aged / Lipopolysaccharides / Adult / Protein Expression / Periodontitis / Cardiovascular Diseases / Human Fibroblasts / Mitochondrial Membrane Potential / Citrate Synthase / Biochemistry and cell biology / PORPHYROMONAS GINGIVALIS

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p53 Mediates Particulate Matter–induced Alveolar Epithelial Cell Mitochondria-regulated Apoptosis

Genetics / Air pollution / Immunohistochemistry / Mitochondria / Apoptosis / Lung Cancer / Humans / Reactive Oxygen Species / Animals / American / Mitochondrial dysfunction / mRna expression levels / Rats / Protein Expression / Particulate Matter / Membrane Potential / Toluene / Mitochondrial Membrane Potential / DNA fragmentation / Lung Cancer / Humans / Reactive Oxygen Species / Animals / American / Mitochondrial dysfunction / mRna expression levels / Rats / Protein Expression / Particulate Matter / Membrane Potential / Toluene / Mitochondrial Membrane Potential / DNA fragmentation

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Mitochondrial bioenergetic deficit precedes Alzheimer\'s pathology in female mouse model of Alzheimer\'s disease

Immunohistochemistry / Oxidative Stress / Mitochondria / Multidisciplinary / Western blotting / Amyloid / Hippocampus / Brain / Humans / Mice / Female / Animals / Temporal Correlation / Amyloid Beta / Lipid peroxidation / Hydrogen Peroxide / Mitochondrial dysfunction / Mouse Model / Time Factors / Neurodegenerative Disease / Alzheimer Disease / Oxygen Consumption / Alcohol Dehydrogenase / Amyloid / Hippocampus / Brain / Humans / Mice / Female / Animals / Temporal Correlation / Amyloid Beta / Lipid peroxidation / Hydrogen Peroxide / Mitochondrial dysfunction / Mouse Model / Time Factors / Neurodegenerative Disease / Alzheimer Disease / Oxygen Consumption / Alcohol Dehydrogenase

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Methylene blue delays cellular senescence and enhances key mitochondrial biochemical pathways

Physiology / Oxidative Stress / Signal Transduction / Humans / Tissue culture / Animals / Male / Cytochrome C / Medical Physiology / Methylene Blue / Cadmium / Enzyme / Hydrogen Peroxide / Mitochondrial dysfunction / Rats / Oxygen / Time Factors / Human Fibroblasts / Alzheimer Disease / Life Span / Oxygen Consumption / Oxidation-Reduction / Oxidants / Biochemistry and cell biology / Thioredoxin reductase / Enzyme Induction / Animals / Male / Cytochrome C / Medical Physiology / Methylene Blue / Cadmium / Enzyme / Hydrogen Peroxide / Mitochondrial dysfunction / Rats / Oxygen / Time Factors / Human Fibroblasts / Alzheimer Disease / Life Span / Oxygen Consumption / Oxidation-Reduction / Oxidants / Biochemistry and cell biology / Thioredoxin reductase / Enzyme Induction

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Transgenic MMP-2 expression induces latent cardiac mitochondrial dysfunction

Mice / Animals / Biochemical / Creatine Kinase / Lipid peroxidation / Mitochondrial dysfunction / Myocardial Infarction / Myocardium / Necrosis / Infarct Size / Ischemia–reperfusion / Transgene Expression / Ischemic Preconditioning / Biochemistry and cell biology / Matrix Metalloproteinase / Mitochondrial dysfunction / Myocardial Infarction / Myocardium / Necrosis / Infarct Size / Ischemia–reperfusion / Transgene Expression / Ischemic Preconditioning / Biochemistry and cell biology / Matrix Metalloproteinase

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439 Mitochondrial Dysfunction Activates CYCLOOXYGENASE-2 Expression in Cultured Normal Human Synoviocytes

Clinical Sciences / Mitochondrial dysfunction

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